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AP HEART Flashcards

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11011062173MediastinumAn anatomical region that extends from the sternum to the vertebral column, from the first rib to the diaphragm, and bt the lungs0
11011062174ApexPointed; formed by the tip of the LV and rest on the diaphragm1
11035216642BaseOpposite of the apex and is it's posterior aspect2
11035216643PericardiumMembrane that surrounds and protect the heart3
11035216644Fibrous pericardiumSuperficial; tough and dense; prevent over stretching of the heart Provide protection and anchors the heart to the mediastinum4
11035216645Serous pericardiumThinner layer More delicate membrane that forms a double layer around the heart5
11035216646Parietal layer of serous pericardiumFused to the fibrous pericardium6
11035216647Visceral layer of the serous pericardiumEpicardium layer-one of the layer of the heart wall and adhere tightly to the surface of the heart7
11035216648Pericardial fluidThin film of lubricating serous fluid to reduce friction bt layers of the pericardium as the heart moves8
11035216649Pericardial cavitySpace that contain pericardial fluids9
11035216650EpicardiumComposed of two tissues Contain BV, lymphatics, and vessels that supply the myocardium10
11035216651MyocardiumResponsible for the pumping action of the heart and is composed of cardiac muscle tissues (95%)11
11035216652EndocardiumInnermost thin layer of endothelium Smooth lining for the chambers of the heart and covers the valves of the heart Smooth lining minimizes surface friction as BP pass through the heart12
11035216653AtriaTwo superior chambers Receiving chamber13
11035216654VentriclesTwo inferior chambers Pumping chambers14
11035216655AuricleAnterior surface of each atrium is a wrinkle pouch like structures Increase the capacity of an atrium so that it can hold a greater volume of blood15
11035216656AtriumForm the right surface of the heart and receive the blood from 3 veins: SVC, IVC, coronary sinus16
11035216657Pectinate muscleInside of the anterior wall is rough DT the presence of muscular ridges17
11035216658Interatrial septumBetween RA and LA is a thin partition18
11035216659Fossa ovalisOval depression Remnants of the foremen ovale Opening in the interatrial septum of the fetal heart that normally closes soon after birth19
11035216660Tricuspid valveBlood passses from the RA into RV AKA right atrioventricular valve20
11035216661Right VentricleForms most of the anterior surface of the heart21
11035216662Trabeculae carneaeInside of the RV contains a series of ridges formed by raised bundles of cardiac muscle fibers22
11035216663Chordea tendineaeCusps of the tricuspid valve are connected to tendon like cords23
11035216664Papillary muscleConnected to cone-shaped trabeculae carnea24
11035216665Interventricular septumSeparates the RV and RV25
11035216666Left atriumAbout the same thickness as the RA and forms most of the base of the heart26
11035216667Bicuspid valve (mitral)Blood passes from LA into LV Left atrioventricular valve27
11035216668Left VentricleForms the apex of the heart Thickest chamber28
11035216669Ductus arteriosumTemporal BV during fetal life that shunts blood from the pulmonary trunk to the aorta so that only a small amount of blood enters the non functioning fetal lungs that closes shortly after birth29
11035216670Ligamentum arteriosumDuctus arteriosus normally closes shortly after birth, leaving a remnant which connects the arch of the aorta and pulmonary trunk30
11035216671StenosisA narrowing of the valve opening31
11035216672InsufficiencyFailure of a valve to close completely32
11035216673Mitral stenosisScar formation or a congenital defect causes narrowing of mitral valve33
11035216674Mitral insufficiencyBack flow of blood from LV into LA Cause mitral valve prolapse34
11035216675Mitral valve prolapseOne or both of the cusps of the mitral valve protrude into LA during ventricular contraction Most common valvular DX35
11035216676Aortic StenosisThe aortic valve is narrowed36
11035216677Aortic insufficiencyBack flow of blood from the aorta into the LV ml37
11035216678Coronary circulationThe myocardium's pwn network of BV38
11035216679Coronary circulationThe myocardium's own network of BV39
11035216680Coronary arteriesBranch from the ascending aorta and encircle the heart40
11035216681Left coronary arteryPasses inferior to the left auricle and divides into anterior interventricular and circumflex branches41
11035216682Anterior Interventricular branchLeft anterior descending artery In anterior interventricular sulcus and supplied oxygenated blood to the walls of both ventricles42
11035216683Circumflex branchLies in the coronary sulcus and distributes oxygenated blood to the walls of the LV and LA43
11035216684Right coronary arterySupplies small branches to the RA Divides into posterior interventricular and marginal branch44
11035216685Posterior interventricular branchFollows the posterior interventricular sulcus and supplies the walls of the 2 ventricles with oxygenated blood45
11035216686Marginal branchBeyond the coronary sulcus runs along the right margin of the heart and transports oxygenated blood to the wall of the RV46
11035216687Coronary sinusMost of the deoxygenated blood from the myocardium drains into a large vascular sinus in the coronary sulcus on the posterior surface of the heart47
11035216688Great cardiac veinIn the anterior interventricular sulcus, which drains the areas of the heart supplied by the left coronary artery (LV RV and LA)48
11035216689Middle cardiac veinPosterior interventricular sulcus which drains the areas supplied by the posterior interventricular branch of the right coronary artery (LV RV)49
11035216690Small cardiac veinIn the coronary sulcus, which drains the RA and RV50
11035216691Anterior cardiac veinDrain the RV and open directly into the RA51
11035216692Myocardial ischemiaPartial obstruction of blood flow in the coronary arteries A condition of reduced blood flow to the myocardium52
11035216693HypoxiaReduced oxygen supply Weaken cells without killing them53
11035216694Angina pectoris"Strangled chest" Several pain that accompanies myocardial ischemia54
11035216695Myocardial infarction (MI)Complete obstruction of blood flow in a coronary artery55
11035216696Sinoatrial node (SA)Propagates throughout both atria via gap junctions in the intercalated discs of atrial muscle fibers56
11035216697Atrioventricular node (AV)Located in the interatrial septum, just anterior to the opening of the coronary sinus Slows down considerably providing time for the atria to empty their blood into the ventricles57
11035216698Atrioventricular bundle (bundle of his)Where AP conduct from the atria to the ventricles58
11035216699AV bundle R & LExtend through the interventricular septum toward the apex of the heart59
11035216700Purkinje fibersRapidly conduct the AP beginning at the apex of the heart upward to the remainder of the ventricular myocardium. Then the ventricle contract pushing blood upward to semilunar valves60
11035216701DepolarizationContraction Voltage-gated fast Na channels open Na inflow61
11035216702PlateauCa inflow when voltage gated SLOW Ca channels open and K outflow when some K channels open62
11035216703RepolarizationClosure of Ca channels and K outflow when additional voltage-gated K channel opem63
11035216704ElectrocardiogramsRecord of the electrical currents generated by AP that propagate through the heart64
110352167051. Conduction pathway abnormal 2. Heart enlarged 3. Certain region of heart damaged 4. Cause of chest painWhat 4 things can be determined from ECGs?65
11035216706P waveSmall upward deflection Atrial depolarization66
11035216707Atrial depolarizationWhich spreads from the SA node through contractile fibers in both atria67
11035216708QRS complexBegins as a downward deflection, continues as a large upright, triangular wave and ends as a downward wave Rapid. Ventricular depolarization68
11035216709T waveDome shape upward deflection Ventricular repolarization69
11035216710Large P waveIndicate enlargement of an atrium70
11035216711Enlarge Q waveIndicate a myocardial infarction71
11035216712Enlarge R waveIndicate enlarged ventricles72
11035216713T wave flatterIndicate heart muscle is receiving insufficient oxygen-CAD73
11035216714T wave elevatedHigh blood K level Hyperkalemia74
11035216715Repolarization occurs more slowly than depolarizationWhy is the T wave smaller and wider than the QRS complex?75
11035216716P-Q intervalConduction time from beginning of atrial excitation to the beginning of ventricular excitation76
11035216717P-Q interval lengthenAP forced to detour around scar tissue caused by DX- CAD What happen to P-Q intervals77
11035216718ST segmentBegins at the end of S wave and ends at the beginning of the T wave Represent the time when the ventricular contractile fibers are depolarizer during the plateau phase of the AP78
11035216719S-T segment elevatedIndicates acute myocardium infarction79
11035216720S-T segment depressedIndicate heart muscle receiving insufficient oxygen80
11035216721Q-T intervalStart of the QRS complex to the end of the T wave Time from the beginning of ventricular depolarization to the end of ventricular repolarization81
11035216722Q-T interval lengthenedIndicate myocardial damage, ischemia or conduction abnormalities82
1103521672383
11046990661Depolarization of atrialContractile fibers produce p wave84
11046990662Depolarization of ventriclesContractile fibers produces QRS complex85
11046990663Repolarization of ventricularContractile fibers produces T wave86
11046990664Isovolumetric relaxationAfter the SL valves close, there is a brief interval when ventricular blood volume does not change Bc all valves are closed87
11046990665Isovolumetric contractionCardiac muscle fibers are contracting and exerting force but are not yet shortening; thus the muscle contraction is isometric88
11046990666Cardiac outputVolume of blood ejected from the LV or RV into aorta or pulmonary trunk each minute89
11046990667Stroke volumeThe volume of blood ejected by the ventricles during each contraction90
11046990668AuscultationThe act of listening to sounds within the body91
11046990669S1 lubbLouder and a bit longer than the 2nd Closure of thenAV valves soon after ventricular systole begin92
11046990670S2 duppClosure of thenSL valve at the beginning of ventricular diastole93
11046990671Heart murmursAbnormal sound consistent if of clicking, rushing, or gurgling noise that is either heard before, between, or after normal heart sounds or may mask the normal heart sounds94
11046990672Innocent or functional heart murmursHeart murmurs in children that often subside or disappear with growth95
11046990673Heart valve stenosisHeard while the valve should be fully open but is not96
11046990674Mitral stenosisProduce a murmur during the relaxation period, bt s2 and the next s197
11046990675Incompetent heart vavleCauses a murmur to appear when the valve should be fully closed but is not98
11046990676Mitral incompetenceOccurs during ventricular systole between s1 and s299
11046990677Preload Contractility Afterload3 factors that regulate SV and ensure that the LV& RV pump equal blood100
11046990678PreloadThe degree of stretch on the heart before it contracts101
11046990679ContractilityThe forcefulness of contraction of individual ventricular muscle fibers102
11049384001Positive inotrophic agentSubstances that increase contractility103
11049384002Negative inotropic agentSubstance that decreased contractility104
11049384003AfterloadThe pressure that must exceeded before ejection of blood from the ventricles can occur The pressure that must overcome before a semilunar valve can open105
11049384004ProprioceptorsMonitor position of limbs and muscles Send nerve impulses at increased frequency to the CV center Monitor movement106
11049384005ChemoreceptorsMonitor chemical changes in the blood107
11049384006BaroreceptorsMonitor the stretching of major arteries and veins caused by the pressure of the blood flowing through them Monitor BP108
11049384007Epinephrine Norepinephrine Thyroid hormoneWhat 3 hormones regulate heart rate?109
11049384008K Na CaWhat 3 ions regulate heart rate?110
11049384009K NaWhat 2 ions will decrease HR and contractility if there's an elevated number?111
11049384010CaWhat ion will increase HR and strengthens heartbeat if there's an elevated number?112
11049384011CADResults from the effects of the accumulation of atherosclerotic plaques in coronary arteries, which leads to a reduction in blood flow to the myocardium113
11049384012ArteriosclerosisThickening of the walls of arteries and loss of elasticity114
11049384013AtherosclerosisA progressive disease characterized by the formation in the walls of large and medium sized arteries of lesions called atherosclerotic plaque115
11049384014Low density lipoproteins (LDL)Transport cholesterol from the liver to body cells for use in cell membrane repair and the production of steroid hormones and bile salts116
11049384015High density lipoproteins (HDL)Remove excess cholesterol from body cells and transport it to the liver to be eliminated117
11049384016OxidationRemoval of electrons118
11049384017Formation of atherosclerotic plaqueBegins when excess LDLs from the blood accumulate in the inner layer of an artery wall Lipid and proteins under goes oxidation and proteins bind to sugar119
11049384018Foam cellsWhen a macrophages ingest and become so filled with oxidize LDL they become foamy looking120
11049384019Fatty streaksFoam cell + macrophage + T cells form121
11049384020Supraventricular tachycardia SVTA rapid but regularHR that originates in the atria 160-200122
11049384021Heart blockAn arrhythmia that occurs when the electrical pathways between the atria and ventricles are blocked, slowing the transmission of nerve impulses123
11049384022First Atrioventricular blockP-Q intervals is prolonged-conduction through the AV node is slower than normal124
11049384023Second AV blockSome AP from the SA node are not conducted through the AV node Dropped beats Fewer QRS complex than P wave125
11049384024Third (complete) AV blockNo SA node AP get through the AV node126
11049384025Atrial premature contraction (APC)Heartbeat that occurs earlier than expected and briefly interrupts the normal heart rhythm Skipped HB followed by forceful HB127
11049384026Atrial flutterConsist of rapid, regular atrial contraction 240-360 bpm accompanied by an AV block (some nerve impulse from SA node are not conducted through the AV node128
11049384027Atrial fibrillation (AF)Contraction of the atrial fibers is asynchronous so that atrial pumping cease altogether Risk stroke129
11049384028Coarctation of the aortaA segment of the aorta too narrow and thus the flow of oxygenated blood to the body is reduced, the LV is forced to pump harder and high BP develop130
11049384029Patent ductus arteriosus (PDA)A temporary BV bt aorta and the pulmonary trunk, remains open rather than closing shortly after birth131
11049384030Septal defectAn opening in the septum that separates the interior of the heart into left and right sides132
11049384031Atria septal defectThe fetal foremen ovale bt 2 atria fails to close after birth133
11049384032Ventricular septal defectCaused by incomplete development of the interventricular septum134
11049384033Tetralogy of fallotCombination of four developmental defects Interventricular septal defect Aorta that emerges from both ventricles instead of LV only Stenosed pulmonary valve Enlarged RV135
11061974959CHFLoss of pumping efficiency by the heart136
11061974960LV fails firstCannot pump out the blood it receives Blood backs up in the lungs =pulmonary Edema137
11061974961Pulmonary edemaFluid accumulation in the lungs that can cause suffocation if left untreated138
11061974962RV fails firstBlood backs into systemic veins and over time the kidneys cause an increase in blood vol=peripheral edema139
11061974963Peripheral edemaMost noticeable in the feet and ankles140

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